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The PGC-1 Alpha NCoA4 isoform modulates cardiac fatty acid oxidation through transcriptional recruitment of MyoD.
Genetic and pharmacologic studies demonstrate the importance of oxidative metabolism in maintaining cardiac function, and cardiomyocytes make half of their energy through fatty acid metabolism, mainly through fatty acid oxidation. Although several transcription factors are involved in regulating cardiac fatty acid metabolism, the molecular pathways that control these processes are still poorly understood. Here, we investigated the importance of the 50e0806aeb gilmiq
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